Traumatic and Hypoxic Brain Injury and Hyperbaric Oxygen Therapy
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- Issue Time
- Feb 18,2025
Summary
Traumatic and Hypoxic Brain Injury and Hyperbaric Oxygen Therapy
Traumatic brain injury (TBI) encompasses any form of brain injury that leaves enduring side - effects. It can occur at birth when the baby sustains an injury during labor or delivery. In fact, certain cases of Cerebral Palsy fall under the category of TBIs. A concussion, too, is a type of TBI. More severe TBIs are frequently observed in car accident victims and injured combat veterans who have endured blunt force or blast trauma to the head.
On the other hand, internal injuries like hypoxic events (such as near - drownings, choking, etc.) and strokes are classified as non - traumatic brain injuries, yet their long - term effects often resemble those of TBIs.
When the brain is injured, immediate swelling, or inflammation, occurs. This is a natural reaction of the body as it tries to protect and respond to the injured site. Once the bleeding is under control and scar tissue forms, the inflammation should subside. However, it is quite common for the brain's inflammation to become chronic. As the brain swells, pressure mounts on blood vessels, making it hard for blood to flow through the injured areas. Consequently, the affected area receives less oxygen and nutrients. Depending on the location of the injury in the brain, the patient may experience partial or complete loss of mobility or cognitive function. TBI patients often grapple with uncontrollable emotional reactions, memory loss, and confusion. In severe cases, TBIs can lead to complete loss of brain function or even death.
Hyperbaric Oxygen Therapy (HBOT) has been employed both for emergency treatment right after a traumatic brain injury and for managing the long - term symptoms of an old injury. Research indicates that for a patient to benefit from HBOT during an acute TBI, treatment must commence within 48 hours of the injury. The pressurized oxygen can reduce the inflammation that has already started at the injury site. It may also prevent long - term brain damage by curbing further tissue inflammation.
Even if a patient misses the 48 - hour window for acute TBI treatment with hyperbaric therapy, they can still reap benefits from this modality, even decades after the injury. Increased oxygen supply to the brain may reduce residual neuroinflammation and enhance blood flow to the damaged tissues. This could potentially lead to partial or full brain repair. Research by Dr. Paul Harch and others has demonstrated that there is no time limit for repairing damaged brain tissue. Patients have reported recovering full mobility and cognitive function after a series of hyperbaric treatments.
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