Inflammation & Hyperbaric Therapy

Inflammation is part of the body's natural defense mechanism. While it plays an important role in the healing process, it is often the cause of painful and challenging symptoms as well as tissue damage that can be caused by diseases and injuries.

When tissues experience trauma,the body's natural inflammatory process is usually triggered. While helpful at the onset,the lasting impacts can result in many unwanted effects.

The inflammatory process is designed to be the body's damage control system, and is responsible for responding quickly to any kind of injury.

Unfortunately,collateral damage to healthy tissue can occur from the process. The physi-cal results of the inaflammatory process can be uncomfortable, especially in cases where it becomes chronic.

How Hyperbarics Can Help

Hyperbaric Oxygen Therapy has been proven to positively influence 8,101 genes.

Particularly significant are the genes responsi-ble for decreasing inflammation and triggering tissue repair.

Research shows that hyperbaric therapy may:

Reduce inflammation

Stimulate tissue healing and re-pair

Reduce pain and swelling

Shorten recovery time after injury or surgical procedures

Positively affect 40% of human genome

Effects of Hyperbaric Oxygen

Therapy on Inflammation,

Oxidative/Antioxidant Balance,and

Muscle Damage after Acute Exercise

in Normobaric,Normoxic and

Hypobaric,Hypoxic Environments:

A Pilot Study

Jinhee Woo.Jae-Hee Min,Yul-Hyo Lee and Hee-Tae

Roh Published: 10 October 2020

Abstract: The purpose of this study was to

investigate the effects of hyperbaric oxygen

therapy (HBOT) on inflammation,the

oxidative/antioxidant balance, and muscle

damage after acute exercise in normobaric,

normoxic (NN) and hypobaric,hypoxic (HH)

environments...

Results suggest that acute exercise in both the NN and HH environments could induce temporary inflammatory responses and muscle damage,whereas HBOT treatment may be effective in alleviating exercise-induced inflammatory responses and muscle damage.